What is the biggest obstacle in the weight loss process?
Two words: appetite
In recent years, “weight loss injections” have become a sensation online, becoming a focal point in discussions about weight loss. They are highly praised for their strong appetite-suppressing effects—once injected into the body, they make food unappealing, allowing for rapid weight loss through extremely low calorie intake. Of course, this effect comes with some side effects.
Many friends have left messages asking us, what is the mechanism of action of this drug?
In fact, the principle is very simple; it utilizes your body’s inherent “weight loss mechanism” to mimic the appetite-controlling hormones naturally secreted by your body.
In other words, there is already a natural form of weight loss injection within you!
However, many people have not learned how to utilize it and have even disrupted this natural mechanism. For those who wish to lose weight but are not overweight, rather than having high expectations for weight loss injections, it would be better to make good use of the inherent weight loss mechanisms in our bodies.
The inherent weight loss mechanism in the body
How does it work?
The weight loss injection that everyone refers to mainly refers to “GLP-1 receptor agonists,” which mimic a hormone called GLP-1 (glucagon-like peptide-1) secreted by your intestines.
GLP-1 acts like your “dietary steward,” managing your appetite and maintaining the balance of weight and blood sugar.
It signals the pancreas to secrete insulin to lower blood sugar levels.
It slows down the motility of the gastrointestinal tract, limiting the speed at which food is absorbed into the bloodstream, while making you feel full.
It informs the brain that you have consumed enough energy and sends signals to stop eating.
Thus, you can contently put down your utensils while GLP-1 is broken down by enzymes, withdrawing from action.
Thanks to this flexible appetite feedback mechanism, you avoid overeating, maintain stable blood sugar levels, and control your weight.
However, for many people, this mechanism has malfunctioned.
Why has the secretion function of GLP-1 gone awry?
Think back, have you ever experienced the following situations:
Others feel satisfied after one bowl of rice, while you need to eat two bowls.
Others can last half a day after a meal, while you can only get by for two hours after one meal.
Others might sometimes have no appetite, while you seem to have never experienced such a feeling.
Others only eat at mealtimes, while you seem to be constantly eating, and it doesn’t affect your appetite even at mealtimes.
……
The situations mentioned above likely indicate that your GLP-1 is no longer functioning normally.
The brain hasn’t received the signals of fullness, thinking you’re still hungry; the gastrointestinal tract hasn’t received the signals to stop, continuously digesting food, like a bottomless pit, never feeling satisfied no matter how much you eat……
Without the “appetite steward” control, we allow our appetites to dominate us, becoming slaves to food.
What follows is a rapid increase in weight, and issues such as insulin resistance and prediabetes soon arise. Studies show that the post-meal secretion of GLP-1 is significantly reduced in obese individuals, those with insulin resistance, or diabetes patients.
Why has this set of appetite management mechanisms failed?
